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. 2020 Mar 13;10(1):2045894019856483. doi: 10.1177/2045894019856483

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© The Author(s) 2020

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Fig. 2. — BMPR2 knockdown in monocytic lineage cells show worsened phenotype following sugen/hypoxia injury in mouse model of PH: (a) RVSP; (b) heart rate; and (c) blood glucose. *P < 0.01 baseline vs. sugen/hypoxia treatment, #P < 0.05 control (LysM Cre mice) vs. BMPR2^KO (LysM Cre BMPR2^KO mice). Black squares represent individual animals.