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. 2020 Jan 30;147(1):37–47. doi: 10.1007/s11060-019-03318-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — p53 mutation promotes cell proliferation, migration and invasion in multifocal primary glioblastoma cell lines in vitro. a Real-time xCelligence analysis of proliferation (represented by cell index) of G52 (red) and G53 (black). b 24 h timepoint analysis of proliferation (represented by cell index) levels between G53 and G52. Error bars represent standard deviations. p < 0.1; *p < 0.02. c Real-time xCelligence analysis of migration (represented by cell index) of G53 and G52. d 24 h timepoint analysis of migration (represented by cell index) levels between G53 (left frontal lesion) and G52 (left thalamic lesion). Error bars represent standard deviations. p < 0.1; p < 0.8. e Real-time xCelligence analysis of invasion (represented by cell index) of G53 (black) and G52 (red). f 24 h timepoint analysis of invasion (represented by cell index) levels between G53 (left frontal lesion) and G52 (left thalamic lesion). Error bars represent standard deviations. p < 0.2; p < 0.1