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. 2020 Mar 16;4:6. doi: 10.1038/s41698-020-0112-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a The OS of the PBRM1-mutant patients was worse than that of those without the mutation in the cohort of ICB-treated patients. b There is marginally significant difference in OS between the PBRM1 mutation subgroup and the PBRM1 wild type subgroup in the cohort of non-ICB-treated patients. No survival difference between PBRM1 mutation types (LOF mutation and non-LOF mutation) were observed in the cohort of ICB-treated (c) and non-ICB-treated (d) patients. OS overall survival, ICB immune checkpoint blockade, LOF loss of function.