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. 2020 Mar 10;14:203. doi: 10.3389/fnins.2020.00203

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Copyright © 2020 Rigby, Gomez and Puglielli.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Molecular mechanisms underlying glial-axon growth cone repulsive signals. MAG, myelin-associated glycoprotein; OMgp, oligodendrocyte-myelin glycoprotein; p75NTR, neutrophin receptor; CRMP2, collapsin response mediator protein family-2; RhoA, Ras family member homolog A; RTK, receptor tyrosine kinase; NG2, neuron-glia antigen-2; CSPG, chondroitin sulfate proteoglycan; RGMa, repulsive guidance molecule A; FAK, focal adhesion kinase; LMO4, LIM domain transcription factor; NgR1, Nogo receptor 1; PirB, paired immunoglobulin-like receptor B; Tim-2, T cell immunoglobulin and mucin domain containing 2; Lrig2, leucine rich repeats and immunoglobulin like domains 2; GAP, GTPase activating protein; PI3K, phosphoinositide 3-kinase; LARG, leukemia-associated Rho guanine nucleotide exchange factor; PKC, protein kinase C; LIMK, LIM domain kinase 1; ROCK, Rho-associated protein kinase; MLC, myosin light chain; ER, endoplasmic reticulum.