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. 2020 Feb 17;2(2):100092. doi: 10.1016/j.jhepr.2020.100092

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© 2020 The Author(s)

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

PMC Copyright notice

Fig. 4 — Markers related to oxidative stress at the end of DHOPE and normothermic reperfusion.

(A) MDA in perfusate at the end of DHOPE (p = 0.312), (B) MDA in liver parenchyma at the end of DHOPE (p = 0.125) and reperfusion (p = 0.604), (C) MDA in perfusate at the end of normothermic reperfusion (p = 0.308), (D) 8-OHdg in the perfusate at the end of normothermic reperfusion (p = 0.172). There were no statistically significant differences between the groups on all parameters (Kruskal-Wallis test). 24 h of DHOPE preservation does not induce oxidative injury compared to shorter preservation times. Shown here are the mean and SEM for n = 6 animals per group. 8-OHdG, 8-hydroxydeoxyguanosine; DHOPE, dual hypothermic oxygenated machine perfusion; MDA, malondialdehyde.