Skip to main content
. 2020 Mar 11;11:142. doi: 10.3389/fgene.2020.00142

Table 3.

In silico pathogenicity analysis of novel RB1 variants.

Patient ID cDNA position Mutation type SIFT PolyPhen-2 PROVEAN Mutation taster ClinPred Human splicing finder MaxEntScan ACMG
RB-0048 c.1100A>G p.N367S Tolerable Benign Tolerable Polymorphism Benign Cryptic donor activated Cryptic donor activated VUS
RB-0100 c.1318G>A p.E440K Tolerable Benign Tolerable Disease-causing Pathogenic VUS
RB-0123 c.1322T>C p.I441T Tolerable Benign Damaging Disease-causing Pathogenic VUS
RB-0134 c.1797T>A p.N599K Damaging Benign Tolerable Disease-causing Pathogenic VUS
RB-0131 c.2260G>A p.V754I Tolerable Benign Tolerable Disease-causing Pathogenic VUS
RB-0143 c.2410A>G p.I804V Tolerable Benign Tolerable Disease-causing Pathogenic VUS
RB-0023 c.264+2T>A Splicing Broken WT Broken splice site P
RB-0087 c.862-2A>T Splicing Broken WT Broken splice site P
RB-0114 c.2490-22_92del Splicing VUS
RB-0065 c.2490-1470 G>A Splicing Produces new acceptor VUS

WT, wild type; P, pathogenic; VUS, variants of uncertain significance.