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. 2020 Mar 18;3:130. doi: 10.1038/s42003-020-0842-3

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — The Swiss IBD cohort study provided colonic or ileal biopsies from Crohn’s disease (CD) or ulcerative colitis (UC) patients which were in remission (quiescent) or with active disease. Healthy patients were recruited at the University Hospital Basel. a CD98hc/SLC3A2 and CD98lc/SLC7A5 expression was determined by qRT-PCR. b Cryosections of inflamed and non-inflamed regions of the same CD or UC patient (patient identification numbers in brackets) were stained for CD98hc and counterstained with NucBlue. Immunofluorescence was carried out with biopsies of five healthy patients, and four UC and four CD patients. c CD98hc fluorescence intensity of staining of biopsies from CD and UC patients. In the figures of the panels (a) and (c), the mean is indicated with each dot representing one patient. The data were analyzed by Mann–Whitney U test; *p ≤ 0.05, **p ≤ 0.01, ***p ≤ 0.001.