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. 2020 Mar 12;11:240. doi: 10.3389/fphar.2020.00240

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Copyright © 2020 Wang, Yang, Wu, Wang, Li, Mei, You and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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DOP attenuated α-SMA and collagen I by modulating inflammatory cytokines in the CCl₄-induced liver fibrosis rat model. (A–C) The mRNA levels of TNF-α, TGF-β, IL-10 in the liver. (D) The protein expression levels of α-SMA in the liver. (E) The mRNA levels of collagen I in the liver. (F–G) An immunohistochemical method was used to detect α-SMA (F) and collagen I (G). The images represented 2–3 different rats in each group (scale bar: 200 μm). Data are presented as the mean ± S.D. (n ≥ 3). ^#p < 0.05, ^###p < 0.001 vs. NC. *p < 0.05, **p < 0.05, and ***p < 0.001 vs. FC.