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. 2020 Mar 19;10:5073. doi: 10.1038/s41598-020-61758-w

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Loss of repression among non-responsive genes in the EARLY UP common abiotic stress regulon. (A) Venn diagram showing the overlap between non-responsive genes in mutants from the EARLY UP stress regulon and the genes already significantly upregulated prior to stress in the mutants, compared to Col-0. The overlap between the two circles represents genes that are non-responsive in mutants due to a loss of repression/increased expression prior to stress exposure. (B) Transcription factor (TF) binding site enrichment analysis was repeated for med16 after non-responsive genes were divided into loss of repression (LR) and loss of activation (LA) according to the Venn diagram. Results were summarized at the level of TF-families. (An extended list of all significantly enriched TFs can be found in Supplemental Fig. S7). (C) Expression levels of representative genes (displaying LR in the med16 mutant prior to stress). Data shown are the average transcripts per million (TPM) ± SD in control conditions (CON) or early cold stress (CS3).