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Schematic diagram of SG formation and dynamic changes during EV-D68 infection. EV-D68 infection induces the formation of typical SGs in which TIA1, HUR, and G3BP1 colocalize early during infection, and the resulting SGs chelate EV-D68 RNA and inhibit viral replication. However, EV-D68 3C protein eliminated the SGs by cleaving the G3BP1 protein as the infection progressed. Thus, SGs are dispersed during the late stage of viral infection.
