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. 2019 Jun 25;32(2):437–447. doi: 10.1007/s12028-019-00748-x

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 8 — Individual P_BTO₂ response to refractory intracranial hypertension expressed relative to changes in ICP (left) and CPP (right) (n = 9). Brain tissue oxygenation response to increases in ICP showed marked variability between patients with some patients showing relatively preserved P_BTO₂ with increasing ICP, some showing precipitous hypoxia, and some showing a paradoxical increase in oxygenation. A similar variability is observed with respect to CPP. This variability highlights the complex nature of the P_BTO₂ signal; it is contributed to by respiratory (FiO₂, PaO₂), hematological (Hb), cardiovascular (cardiac output, cerebral blood flow) and metabolic factors (brain metabolism)