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. 2020 Mar 20;21:71. doi: 10.1186/s12931-020-1331-4

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© The Author(s). 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Effect of MSC-exo MCT-induced pulmonary artery pressure, vascular remodeling and right ventricle hypertrophy. a Comparative analysis of RVSP and RV/(LV + S) ratios (b) Morphology and histology of the heart were observed by hematoxylin and eosin (HE) and Masson’s staining. c Comparative analysis of fibrosis and cardiac output. d Pulmonary arterioles wall thickness and fibrosis were observed by HE and Masson’s staining. e Comparative analysis of the percent of muscular artery (MT%) and optical density (OD). n = 5 rats per group; P < 0.05, t-test; the data are present as mean ± SD; ^*MCT or ^*MSC-CM vs. control; ^#MSC-exo vs. MCT group; scale bar = 100 μm