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. 2020 Mar 21;20:106. doi: 10.1186/s12883-020-01681-9

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Gross and microscopic pathology. a Gross pathology of aortic arch revealing extensive atheromas (black arrows). b Histopathology of the anterior cerebral artery. There is intimal thickening underlying the internal elastic lamina (blue arrow); the media and adventitia are distorted by fibrosis (blue dashed arrow). A recently-formed fibrin thrombus (black arrow) is adherent to a chronic and organized atherosclerotic plaque (red arrow). c Cross-sectional view of the left anterior descending coronary artery containing an intramural calcified nodule (black arrow). d Cross-sectional view of the basilar artery wall, demonstrating architectural distortion with cholesterol clefts (black arrow) and microcalcifications. e Left anterior descending artery with thickening of the intima and inflammatory infiltrate including T lymphocytes (red arrows) and macrophages (black arrows)