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. 2020 Feb 7;19(4):2588–2596. doi: 10.3892/etm.2020.8497

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Copyright: © Li et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — CD36 and ATG7 reverse the antioxidant effect of CD5L on hepatocytes. (A-D) Hepatocytes transfected with siRNA-CD36, siRNA-ATG7 or siRNA-NT and cultured under I/R conditions with or without CD5L. Hepatocytes without any treatment were used as controls. (A) Intracellular ROS production was analyzed by fluorescence spectrophotometry. Commercial kits were used to determine the levels of (B) SOD, (C) CAT and (D) GSH-Px activities in hepatocytes. Data represent the mean ± SD from three independent experiments. ^*P<0.05 vs. control; ^▲P<0.05 vs. I/R + CD5L; ^○P<0.05 vs. I/R + CD5L + siRNA-NT. DFS, dual fluorescent staining; I/R, ischemia/reperfusion; CD5L, CD5-like; CD36, cluster of differentiation 36; ATG7, autophagy-related 7; siRNA, small interfering RNA; NT, non-targeting; ROS, reactive oxygen species; SOD, superoxide dismutase; CAT, catalase; GHS-Px, glutathione peroxidase.