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. 2020 Mar 20;18(1):1559325820913786. doi: 10.1177/1559325820913786

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 2. — LINC01614 was highly regulated in H9c2 cells exposed to hypoxia/reoxygenation (H/R) stimulation and downregulated LINC01614 attenuated myocardial injury caused by H/R treatment. A, LINC01614 expression level was determined by quantitative reverse transcription polymerase chain reaction in sham and H/R groups, **P < .01 versus sham group. B, The detection of silence efficiency of LINC01614, **P < .01 versus si-con group. C, A Cell Counting Kit-8 was used to examine the H9c2 cell viability, **P < .01 versus sham group, ^## P < .01 versus the H/R + si-con group.