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. 2020 Mar 20;18(1):1559325820913786. doi: 10.1177/1559325820913786

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© The Author(s) 2020

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 4. — MiR-138-5p was directly targeted by LINC01614. A, Predicted binding site between miR-138-5p and LINC01614. B, Luciferase activity was measured to verify the correlation between miR-138-5p and LINC01614, **P < .01 versus miR-138-5p mimic negative control (NC). C, Expression level of miR-138-5p was detected after hypoxia/reoxygenation (H/R) treatment and/or si-LINC01614 transfection, **P < .01 versus sham group, ^## P < .01 versus the H/R + si-con group.