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. 2020 Mar 17;11:236. doi: 10.3389/fphar.2020.00236

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Copyright © 2020 Wang, Li, Tu, Chen, Zeng and Jiang

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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TGs attenuate neuronal injury after I/R-treated rats. (A) Nissl staining in the penumbra of ischemic area of I/R rats. Normal neurons have normal morphology with clear nucleolus, abundant cytoplasm, and intact structure (red arrow). Abnormal neurons are shrunken and deep stained (yellow arrow), while a greater increase of intact cells is evident in the TGs group compared with the MOD group. a–f are NOR, MOD, EDI, TGs, PSs and OSs groups, respectively. (B) Quantitative analysis of intact cells in penumbra of ischemic area at 14 days after treatment. *P < 0.05 vs NOR, ^# P < 0.05 vs MOD.