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. 2020 Mar 24;10:5306. doi: 10.1038/s41598-020-62084-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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MAYV infection is associated with acute production of inflammatory mediators. 6-week-old BALB/c mice were inoculated or not with wt MAYV (2 × 10⁵ PFU/50 μL, i.pl.) and levels of (a) IL1-β; (b) IL-6; (c) IFN-γ; (d) TNF-α; (e) VEGF; (f) IL-17; (g) IL-10; (h) CXCL-1; (i) CCL2; (j) CCL3; (k) CCL4 and (l) CCL5 in the spleen were analyzed by ELISA in different time-points. Results are expressed as pg/100 mg of spleen. Data is representative of two experiments expressed as mean ± SEM. *p < 0.05 when compared to control uninfected mice (MOCK), as assessed by one-way ANOVA followed by Newman-Keuls post-test.