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. 2020 Mar 19;13:2347–2355. doi: 10.2147/OTT.S241818

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© 2020 Zhao et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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Overexpression of lncRNA FOXP4-AS1 promotes CC cell proliferation, migration, and invasion in vitro.

Notes: (A) FOXP4-AS1 was overexpression in CC cells by transfection with pFOXP4-AS1. (B) Effects of FOXP4-AS1 overexpression on CC cell proliferation were measured by CCK-8 assay. (C) Roles of FOXP4-AS1 overexpression on CC cell migration were analyzed with wound-healing assay. (D) Effects of FOXP4-AS1 overexpression on CC cell invasion were measured by transwell invasion assay. **P < 0.01, *** P < 0.001.

Abbreviations: lncRNA, long non-coding RNA; FOXP4-AS1, forkhead box P4 antisense RNA 1; CC, cervical cancer; CCK-8, cell counting kit-8.