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. 2020 Mar 26;11:136. doi: 10.1186/s13287-020-01637-3

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — CS-IGF-1C hydrogel augmented the proangiogenic capacity of hP-MSCs. a Scratch wound healing assay of HUVECs treated with CS-IGF-1C hydrogel for 0 and 12 h. *P < 0.05 versus PBS; scale bar represented 200 μm. b The migration ratio statistics of HUVECs in each group. c Representative images of myocardium sections stained for CD31 (red) at day 14. Scale bar, 100 μm. d Quantitative analysis of CD31 immunostaining in each group. The number of capillary vessels was counted in three randomly selected areas. Data are expressed as mean ± SEM. *P < 0.05 versus PBS; **P < 0.01 versus PBS; ^#P < 0.05 versus PMSC. e Expression levels of VEGF by RT-PCR in each group on day 14. Data are expressed as mean ± SEM. *P < 0.05 versus PBS; **P < 0.01 versus PBS