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. 2020 Jan 15;318(3):C536–C541. doi: 10.1152/ajpcell.00568.2019

Fig. 2.

Fig. 2.

Vitamin D receptor-knockdown (VDR-KD) myoblasts display reduced mitochondrial respiration compared with control. A: oxygen consumption rate (OCR) during analysis of respiratory control in control and VDR-KD myoblasts. B: respiratory control parameters from control and VDR-KD myoblasts. C: OCR during analysis of respiratory control in control and VDR-KD myotubes. D: respiratory control parameters from control and VDR-KD myotubes. E: estimations of total ATP production (ATPTotal), oxidative phosphorylation (ATPOx), and glycolysis (ATPGlyc) in control and VDR-KD myoblasts. F: mitochondrial membrane potential assessed via TMRE (tetramethylrhodamine ethyl ester) fluorescence in control and VDR-KD myoblasts. *P < 0.05, **P < 0.005, independent t test. Data are means ± SD (AE: n = 9–10 wells/group. F: n = 5 wells/group).