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. Author manuscript; available in PMC: 2021 Jun 1.
Published in final edited form as: J Comp Neurol. 2019 Dec 16;528(8):1436–1456. doi: 10.1002/cne.24834

Fig. 1.

Fig. 1

Vesicular GABAergic Transporter (vGAT) and Glutamic acid decarboxylase 67 (GAD67) immunoreactivity (IR) in the ventral anterior parvocellular part (VApc) and centromedian (CM) thalamic nuclei. Sagittal sections of the VApc (a-d) and CM (f-k) in control (n=6) and MPTP-treated (n=4) animals processed for vGAT-IR, GAD67-IR, and calbindin. (e,l) Intensity of vGAT-IR and GAD-67 IR in the VApc and CM of control and MPTP-treated monkeys, expressed as ratio of staining intensity (e) VApc/medial geniculate nucleus (MGN) or (I) CM/ventral posteromedial nucleus (VPM). GPe, GPi Globus pallidus external, internal (respectively), RTN reticular thalamic nucleus, IC internal capsule, VL ventral lateral thalamic nucleus. Data are expressed as mean± SEM. Scale bar in a (applies to b-k) = 4mm.