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. 2020 Mar 30;21:265. doi: 10.1186/s12864-020-6684-z

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© The Author(s). 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Comparison of the number of HepG2 genes that were commonly expressed or uniquely expressed for each treatment condition and the untreated cells. a Comparison of V1-treated HepG2 cells and untreated HepG2 cells. b Comparison of V3-treated HepG2 and untreated HepG2 cells. c Comparison of V5-treated HepG2 cells and untreated HepG2 cells. d Four-way comparison of the number of HepG2 genes expressed in all three treatment conditions and the untreated cells. The segment marked blue represent the common gene expression signature for the treated and untreated HepG2 cells. Expressed genes were detected using a detection p value threshold of p < 0.05. e 26 genes related to the blue segment in (d) of expression in all treatments but not in control. f 123 genes expressed exclusively in V3 related to the green segment in (d)