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. 2020 Mar 30;39:55. doi: 10.1186/s13046-020-01555-5

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — circFOXM1 promotes cell proliferation via the miR-614/FAM83D axis. a-b RNA pull-down assay was used for the detection of circFOXM1 and FAM83D by 3′-end biotinylated miR-614. c-d qRT-PCR analysis for mRNA levels of FAM83D in NSCLC cells with circFOXM1 overexpressing or silencing. e Western blot analysis for protein levels of FAM83D in NSCLC cells with circFOXM1 overexpressing or silencing. f Western blot analysis for downstream proteins of FAM83D after transfection with control, circFOXM1, circFOXM1 + NC, or circFOXM1 + sh-FAM83D. g-h Cell proliferation analysis for NSCLC cells transfected with control, circFOXM1, circFOXM1 + NC, or circFOXM1 + sh-FAM83D by CCK-8 assay. *P < 0.05; **P < 0.01