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. 2004 Dec;19(12):3180–3182. doi: 10.1093/ndt/gfh436

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Nephrol Dial Transplant Vol. 19 No. 12 © ERA-EDTA 2004; all rights reserved

This article is made available via the PMC Open Access Subset for unrestricted re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the COVID-19 pandemic or until permissions are revoked in writing. Upon expiration of these permissions, PMC is granted a perpetual license to make this article available via PMC and Europe PMC, consistent with existing copyright protections.

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Fig. 1. — (a) The kidney from case 1. The renal tubule showed prominent cytoplasmic wasting pigment deposition with lymphocytes. The casts in the tubules (arrow) are positive for myoglobin (immunohistochemical stain, 200×). (b) The skeletal muscles from case 2. Myoid giant cells (white arrow) and interstitial ecstatic lymphovascular structures with inflammatory cell infiltration are evident (HE stain, 100×). (c) The kidney from case 2. There are several Mallory body-like casts (arrowhead) in dilated lumens with some sloughed and flattened renal tubular epithelial cells, indicating rhabdomyolysis and acute tubular necrosis. Interstitial infiltration of mononuclear cells is evident. There was a mild increase in mesangial cellularity and matrix (HE, 200×). (d) The glomerulus of case 2 (electron microscopy). Intact basement membrane surrounded by podocytes (×11 500). There was no immune complex deposition or virus particles.