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. 2020 Mar 9;43(5):1491–1502. doi: 10.3892/or.2020.7537

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Flow cytometric analysis of (A) FRO cells incubated with (17-AAG+Torin2)@MSNs or (17-AAG+Torin2)@MSNs-anti-VEGFR2 ab for 0.5 h, respectively. (B) FRO cells incubated with (17-AAG+Torin2)@MSNs or (17-AAG+Torin2)@MSNs-anti-VEGFR2 ab for 3 h, respectively. (C) FRO cells incubated with (17-AAG+Torin2)@MSNs or (17-AAG+Torin2)@MSNs-anti-VEGFR2 ab for 8 h, respectively. 17-AAG, 17-allylamino-17-demethoxy-geldanamycin; Torin2 9-(6-aminopyridin-3-yl)-1-(3-(trifluoromethyl)phenyl)benzo[h][1,6]naphthyridin-2(1H)-one; VEGFR2, vascular endothelial growth factor receptor 2.