Table 1.

Summary of historical and current HRV groupings

	Human rhinovirus A antiviral group B		Human rhinovirus B antiviral group A
1M	34H	64H	3H
2M	36	65	4
7H	38H	66	5
8H	39	67	6H
9H	40H	68	14H
10H	41	71	17H
11H	43	73	26H
12H	44	74	27H
13H	45	75	35
15H	46	76	37
16H	47	77	42
18H	49H	78	48
19H	50	80	52
20H	51H	81	69
21H	53	82	70
22H	54	85	72
23H	55	87	79
24H	56	88	83
25H	57	89	84
28H	58	90	86
29M	59	94	91
30M	60	95	92
31M	61	96	93
32	62	98	97
33H	63	100	99
		HANKS

Minor group HRV strains are underlined, major group are shown in bold. M (only found in HRV A; propensity for monkey cells) and H strains (located in both species; grew in human cell cultures) (Gwaltney and Jordan, 1964, Stott et al., 1969, Rosenbaum et al., 1971, Tyrrell and Parsons, 1960, Tyrrell and Bynoe, 1961, Ketler et al., 1962, Taylor-Robinson and Tyrrell, 1962, Cooney and Kenny, 1977, Bloom et al., 1963) are indicated with superscripts and generally correlate with receptor usage (Macnaughton, 1982). Assignment of some strains to this sub-classification may also have been influenced by the viral load in the inoculum (Douglas et al., 1966a, Hamre, 1967). The M and H terminology was abandoned in favour of a more streamlined sequential numbering system (Taylor-Robinson and Tyrrell, 1962). HRV strains were later divided into the major and minor groups defined by receptor tropism (Abraham and Colonno, 1984, Colonno et al., 1986). Two ‘antiviral groups’ (A and B) were defined by their susceptibility to antiviral molecules, reflected by the amino acid sequence of the interacting regions (Andries et al., 1990). HRV-87 is included in this table although it is a variant of HEV-68 (Ishiko et al., 2002), despite its apparent sensitivity to acid (Uncapher et al., 1991). HRV-Hanks and HRV-21 may be the same serotype as may HRV-8 and HRV-95 (Ledford et al., 2004).