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. 2006 Apr 1;193(7):945–953. doi: 10.1086/500954

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© 2006 by the Infectious Diseases Society of America

This article is made available via the PMC Open Access Subset for unrestricted re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the COVID-19 pandemic or until permissions are revoked in writing. Upon expiration of these permissions, PMC is granted a perpetual license to make this article available via PMC and Europe PMC, consistent with existing copyright protections.

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H9N2/G1 and H5N1/97 infection sensitizing Jurkat T cells to induction of apoptosis by tumor necrosis factor (TNF)–α, TNF-related apoptosis-inducing ligand (TRAIL), and Fas ligand (FasL). After 1 h of virus adsorption, H9N2/G1- and H5N1/97-infected Jurkat T cells were cultured in presence of exogenous TNF-α (500 ng/mL), TRAIL (10 ng/mL), or FasL (10 ng/mL), respectively, for another 24-h culture. A significantly higher percentage of apoptotic cells was observed (P<.05). Data are the mean ± SE from 3 independent experiments