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. 2020 Mar 13;21(5):2095–2102. doi: 10.3892/mmr.2020.11026

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Copyright: © Deng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Effect of β-asarone on autophagy in Alzheimer's disease model mice. Transmission electron microscopy (×30,000 magnification) demonstrated that autophagosome formation was barely observed in the normal group. In addition, there were more autophagosomes in the model- and rapamycin-treated groups compared with the 3-MA and β-asarone treated groups. A, Normal group; B, model group; C, low-dose β-asarone group; D, medium-dose β-asarone group; E, high-dose β-asarone group; F, 3-MA-treated group; and G, the rapamycin-treated group. Data are presented as the mean ± SD of 3 mice. Black arrows indicate autophagosomes. ^**P<0.01 vs. normal group; ^##P<0.01 vs. model group; ^$P<0.05 and ^$$P<0.01 vs. rapamycin-treated group; ^¤¤P<0.01 vs. 3-MA-treated group. 3-MA, 3-methyladenine.