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. 2020 Mar 4;56(5):1083–1092. doi: 10.3892/ijo.2020.5003

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Copyright: © Zhao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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FOXF1 promotes the self-renewal capability and tumorigenesis capacity. (A) Overexpression of FOXF1 increased the self-renewal capability of A549 and H1299 cells, and knockdown of FOXF1 decreased self-renewal capability in A549/DDP cells. (n=3); ^*P<0.05 (FOXF1 plasmids compared to control plasmids, FOXF1 shRNAs compared to control shRNAs) (B) Overexpression of FOXF1 in A549 cells increased the volumes and weights of xenograft tumors. (n=4); ^*P<0.05 (FOXF1 plasmids compared to control plasmids). FOXF1, forkhead box F1.