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. 2020 Jan 3;22(4):758–766. doi: 10.1038/s41436-019-0733-5

Table 1.

Demographic, environmental, and reproductive information on study participants.

All PM FXPOI No FXPOI
N 355 87 168
Age at interview 47.4 ± 12.5 46.2 ± 10.6 53.5 ± 8.8a

Mean ± SD

(min–max)

(19–93) (26–72) (37–80)
Race
  % White 90.4 93.1 88.7
  % Black 3.7 2.3 4.8
  % Hispanic 3.9 2.3 4.8
  % Other 2.0 2.3 1.7
Body mass index (BMI)

27.4 ± 6.8

(17.4–63.4)

27.6 ± 6.8

(17.5–49.9)

28.0 ± 6.4

(18.2–55.0)

% Ever smoked 27.7 24.1 29.3
Number of children 1.7 ± 1.3 1.6 ± 1.2 2.0 ± 1.1b

Mean ± SD

(Min–max)

(0–8) (0–5) (0–6)
Number of children with FXS 0.8 ± 0.8 0.7 ± 0.8 0.8 ± 0.7

Mean ± SD

(Min–max)

(0–3) (0–2) (0–3)
% Satisfied with number of children 78.7 65.5 88.1c
Number of conditions: mean ± SD; median; mode (min–max)

4.0 ± 3.5; 3; 1

(0–16)

4.4 ± 4.0; 3; 1

(0–16)

3.9 ± 3.4; 3; 0

(0–15)

Repeat size

  Mean ± SD

(Min–max)

91.9 ± 19.4

(56–190)

89.6 ± 14.2

(56–140)

91.7 ± 20.7

(56–190)

  % 55–79 25.0% 19.8% 28.1%d
  % 80–100 49.4% 61.6% 44.5%
  % 101–200 25.6% 18.6% 27.4%

FXPOI fragile X–associated primary ovarian insufficiency, FXS fragile X syndrome, PM premutation.

ap < 0.0001 using a t-test to compare means among women with and without FXPOI.

bp < 0.05 using a t-test to compare means among women with and without FXPOI.

cp< 0.0001 using chi-square analysis to compare frequencies among women with and without FXPOI.

dp < 0.05 using chi-square analysis to compare frequencies among women with and without FXPOI.