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Immune escape of lymphoblasts possibly due to enhanced sialylation, O-acetylation of glycoproteins or disialo gangliosides GD3 [156–158], SOAT [146], and reduced membrane bound sialidase (Neu 3) [159] in ALL. Subclass switching of anti-Neu5,9Ac2GPs antibodies from IgG1 to IgG2, modulation of Fc-glycosylation, and weakening a few Fc-glycosylation-sensitive effector functions seem to be responsible for evading the host’s immune response [160]
