Supplementation of Se upregulates the expression of selenoproteins, which mediate the shift of the ARA pathway to produce more anti-inflammatory PGD2 and its downstream CyPG metabolites, while decreasing the levels of pro-inflammatory PGE2 and TXA2 via the regulation of PG synthases (mPGES-1, TXS, and H-PGDS) as well as two transcription factors, i.e. NF-ĸB and PPARγ.