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. 2015 Apr 30:465–514. doi: 10.1007/978-94-017-9882-2_22

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© Springer Science+Business Media Dordrecht and People's Medical Publishing House 2015

This article is made available via the PMC Open Access Subset for unrestricted research re-use and secondary analysis in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the World Health Organization (WHO) declaration of COVID-19 as a global pandemic.

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Fig. 22.31 — Pathology of influenza A (H1N1). (a–e) Extended and congested hepatic sinus, cholestasis of central hepatocytes, bullous fatty degeneration of hepatocytes, and infiltration of inflammatory cells around portal areas (H&E (a–d) ×10, (e) ×20). (f, g) Dilated and congested splenic sinus, atrophic splenic corpuscles, and decreased lymphocytes (H&E ×10, ×20). (h) Vascular hemorrhage of intestinal mucosa (H&E ×10)

Fig. 22.31 — Pathology of influenza A (H1N1). (a–e) Extended and congested hepatic sinus, cholestasis of central hepatocytes, bullous fatty degeneration of hepatocytes, and infiltration of inflammatory cells around portal areas (H&E (a–d) ×10, (e) ×20). (f, g) Dilated and congested splenic sinus, atrophic splenic corpuscles, and decreased lymphocytes (H&E ×10, ×20). (h) Vascular hemorrhage of intestinal mucosa (H&E ×10)