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. 2020 Apr 5;8:42. doi: 10.1186/s40478-020-00916-7

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — ANXA2- and OSMR-mediated regulation of tumor growth and phenotypic transition in a mouse xenograft model of GBM. a Kaplan–Meier survival curves of BALB/c-nu/nu mice injected intracranially with MGG23 cells expressing the indicated shRNAs (n = 6). b qRT-PCR using FFPE tissue sections of intracranial xenografts. c, d Immunohistochemical staining of human leukocyte antigen (HLA), Ki67, and CD31 in MGG23 tumors excised on day 91 after tumor cell injection (n = 5). Representative images (c) and quantification (d) of staining. Yellow dotted lines outline contralateral white matter tracts. Data are shown as the mean ± SEM. *P < 0.05, **P < 0.01, ***P < 0.001 by the log-rank test (a) or one-way ANOVA with Bonferroni’s post hoc test (c)