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. 2020 Mar 23;21(6):2228. doi: 10.3390/ijms21062228

Figure 1.

Figure 1

Human idiopathic pulmonary fibrosis (IPF) primary lung-myofibroblasts, compared to normal-, primary-lung myofibroblasts, decrease miR-34a levels and show an inverse correlation with FLICE-like inhibitory protein (FLIP) expression (A) Graphical presentation of FLIP protein expression determined by optical densities (OD) in Western blot (WB), and (B) qPCR miR-34a expression levels (calculated as 2−∆∆Ct) in normal (NL) vs. fibrotic (IPF) lung myofibroblasts. n = 5–6. (C) qPCR for miR-34b and (D) miR-34c in NL and IPF human lung myofibroblasts. Error bars represent mean ± SD; circles represent individual data points. * p < 0.01.