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CIKs integrate RLK-mediated pathways to maintain stem cell homeostasis in the SAM. CIKs redundantly interact with CLV1, CLV2/CRN, and RPK2 to transduce the CLV3 signal into the nucleus while suppressing the expression of WUS. CIKs can at least be phosphorylated by CLV1 and RPK2 for signaling, according to the results from phosphorylation assays (Cui et al., 2018; Hu et al., 2018). Question marks indicate that no physical interaction between CLV3 and RPK2 or CLV2 has been confirmed.
