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. 2020 Apr 8;11(4):227. doi: 10.1038/s41419-020-2416-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Expression levels of miR-107 in HOTAIRM1-overexpressing B-CPAP and TPC-1 cells transfected with miR-NC or miR-107 mimics, assessed by qRT-PCR (n = 3, **p < 0.001). b Micro-RNA-107 mimics reversed the HOTAIRM1-induced inhibition of cell proliferation (n = 3, **p < 0.001). c miRNA-107 mimics reversed the HOTAIRM1-induced cell apoptosis (N = 3, **p < 0.001). d, e miRNA-107 mimics restored the migration and invasion ability impaired by HOTAIRM1 in B-CPAP (D) and TPC-1 (E) cells (n = 3, **p < 0.001). Scale bar: 100 μm. f Migration was evaluated by wound healing assays in B-CPAP and TPC-1 cells stably overexpressing HOTAIRM1 and transfected with miR-NC or miR-107 mimics (n = 3, **p < 0.001).