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. 2020 Apr 2;14:223. doi: 10.3389/fnins.2020.00223

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Copyright © 2020 Zhao, Hou, Feng, Shen, Nan, Zhang, Wang, Ma and Feng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effects of Vinp on infarction size, neurological deficits, brain water content, and oxidative stress in rats following MCAO. (A) Neurological deficit analysis (n = 16 in each group). (B) Brain water content analysis (n = 3 in each group). (C) Representative images of cerebral infarction after ischemia for 2 h and reperfusion for 12 h in rat brains by TTC staining. (D) Analysis of infarct size calculated as a percent relative to total cerebral volume (n = 3 in each group). (E) Analysis of SOD activity (n = 5 in each group). (F) Analysis of MDA content (n = 5 in each group). *P < 0.05, **P < 0.01, ***P < 0.001 using one-way ANOVA followed by Tukey’s post hoc tests. Vinp: Vinpocetine; LY: LY294002; I/R: ischemia/reperfusion; MCAO: middle cerebral artery occlusion; TTC: 2,3,5-triphenyltetrazolium chloride.