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. 2020 Apr 2;14:223. doi: 10.3389/fnins.2020.00223

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Copyright © 2020 Zhao, Hou, Feng, Shen, Nan, Zhang, Wang, Ma and Feng.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effects of Vinp on inflammation and apoptosis-related proteins, and the expression of Cx43, p-Cx43, AKT, and p-AKT in the rat cerebral cortex after I/R injury. (A) Representative western blot images and quantification of proteins related to inflammation. (B) Representative western blot images and quantification of proteins related to apoptosis. (C) Representative western blot images of Cx43 and p-Cx43, and quantification of Cx43, p-Cx43, and p-Cx43/Cx43. (D) Representative western blot images of AKT and p-AKT, and quantification of AKT, p-AKT and p-AKT/AKT. Data are shown as the mean ± SEM (n = 5 in each group). **P < 0.01, ***P < 0.001 using one-way ANOVA followed by Tukey’s post hoc tests. Vinp: Vinpocetine; LY: LY294002; I/R: ischemia/reperfusion.