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. 2020 Mar 23;58(1):239–246. doi: 10.1080/13880209.2020.1739719

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© 2020 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — CNE decreased the liver index and the levels of AST, ALT, and MDA in rats with CCl₄-induced ALI. Liver index (A), body:liver weight ratio in rats; the MDA (B), AST (C), and ALT (D) levels in liver and serum. The data are shown as mean ± SD, n = 10. #p < 0.05 compared to the control group; *p < 0.05 compared to negative group, for all figures. CNE attenuated the histopathological changes induced by CCl₄ in rats with ALI (E). CNE prevented inflammation (), necrosis (), steatosis (), and ballooning degeneration () in ALI rats. The HE staining of liver sections obtained from (a) control group; (b) negative group; (c) positive group; (d)–(f) CNE groups treated with 40, 80, and 160 mg/kg, respectively. Scale bar = 20 µm.

Inline graphic — CNE decreased the liver index and the levels of AST, ALT, and MDA in rats with CCl₄-induced ALI. Liver index (A), body:liver weight ratio in rats; the MDA (B), AST (C), and ALT (D) levels in liver and serum. The data are shown as mean ± SD, n = 10. #p < 0.05 compared to the control group; *p < 0.05 compared to negative group, for all figures. CNE attenuated the histopathological changes induced by CCl₄ in rats with ALI (E). CNE prevented inflammation (), necrosis (), steatosis (), and ballooning degeneration () in ALI rats. The HE staining of liver sections obtained from (a) control group; (b) negative group; (c) positive group; (d)–(f) CNE groups treated with 40, 80, and 160 mg/kg, respectively. Scale bar = 20 µm.