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. 2020 Apr 7;11:270. doi: 10.3389/fphar.2020.00270

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Copyright © 2020 Liu, Han, Gao, Gao, Yao and Liu

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Upregulated miR-155, downregulated SOCS1, and activated MAPK signaling pathway are observed in the liver tissue of AH rats. (A) mRNA expression of miR-155, SOCS1, p38, ERK1/2, and JNK in rat liver tissues. (B, C) Protein expression of SOCS1, p38, ERK1/2, and JNK in rat liver tissue. *p < 0.05 AH vs. normal hepatic stellate cells. The measurement data are expressed as mean ± standard deviation and compared by an unpaired t-test (n = 8). SOCS1, suppressor of cytokine signaling 1; MAPK, mitogen-activated protein kinase; AH, alcoholic hepatitis; RNA, ribonucleic acid; ERK, extracellular regulated protein kinases; JNK, Jun N-terminal kinase.