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. 2020 Apr 13;48(4):0300060520903652. doi: 10.1177/0300060520903652

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© The Author(s) 2020

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 4. — L02 (a) and HepG2 (b) cells were treated with 0, 2.5, 5.0, 7.5, and 10 µg/mL TiO₂, and the cell cycle distribution was then examined by flow cytometry. TiO₂ caused release of ROS in liver cancer cells. Cells incubated with 0 µg/mL TiO₂ were used as a control. ***P < 0.001 vs. 0 µg/mL (control). DCFDA, 2’,7’-dichlorofluorescin diacetate.