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. 2020 Mar 2;52(3):409–422. doi: 10.1038/s12276-020-0390-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a GSEA analysis showing the positive correlation between PROM2 expression and gene sets relevant to the Akt signaling pathway. b Immunoblotting assays demonstrating the expression level of the indicated proteins in AsPC-1 cells with different expression level of PROM2. c Luminescence signaling (left) and tumor weight (right) of the tumors formed by AsPC-1/PROM2 after treatment with vehicle or the Akt inhibitor (MK-2206) (*P < 0.05). d Representative images (left) and quantification (right) of TUNEL and active caspase-3 in tumors with the indicated treatment (*P < 0.05).