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. 2020 Apr 14;10:6265. doi: 10.1038/s41598-020-63284-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Immunocytochemistry of fibronectin, COL1A1, and αSMA in hTMs exposed to the conditioned medium. The left panels show cells that were stained with DAPI. The middle panels show cells stained for fibronectin, COL1A1, and αSMA. The right panels show the merged image. The expression of fibronectin, COL1A1, and αSMA increased post-infection, and were attenuated with each inhibitor or antagonist application. Bar, 200 μm.