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. 2020 Feb 18;122(8):1185–1193. doi: 10.1038/s41416-020-0757-2

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a–d Flt3L-induced BM DCs from control (Xcr1^+/venus, filled circles) or XCR1-deficient (Xcr1^venus/venus, open circles) mice were incubated with indicated concentrations of XCL1-OT-I (a, b) or OVA protein (c, d) for 6 h, and analysed by a flow cytometer. Percentages of CD11c⁺CD24^highCD11b^int cells (cDC1) (a, c) and CD11c⁺CD24^intCD11b^high cells (cDC2) (b, d) expressing OT-I/H-2K^b complex are shown. Similar data were obtained from two independent experiments. *p < 0.05.