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. 2019 Jun 19;70(9):1855–1864. doi: 10.1093/cid/ciz526

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© The Author(s) 2019. Published by Oxford University Press for the Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — Maximum likelihood SNV tree for all ST-26 isolates. SNVs were identified from mapping reads to a de novo assembled study isolate genome (ldn_gos_2_520). Samples are highlighted based on inclusion in sequence clusters. The tree is annotated with the presence (black) and absence (white) of accessory genes, as well as the presence of AMR-associated genes and mutations. This includes the presence of a functional erm(41) gene conferring inducible resistance to macrolides; the presence of 2 rrl mutations conferring high-level macrolide resistance; and the presence of a mutation in rrs conferring high-level amikacin resistance. The scale bar represents the number of SNVs and the node bootstrap scores below are shown if below 75. Abbreviations: AMR, antimicrobial resistance; SNV, single nucleotide variant.