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. 2020 Apr 15;19:75. doi: 10.1186/s12944-020-01260-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Association of HDL functions with acute coronary syndrome. Odds ratios per 1-SD increase their respective 95% confidence intervals (CIs) and p values are shown for acute coronary syndrome. Dots represent odds ratios, and error bars indicate 95% confidence intervals. Logistic regression model 1 indicates unadjusted odds ratio, model 2 adjusted for age, gender, BMI, smoking status, diabetes, hypertension and LDL-C. In model 3, additional adjustment for HDL-C was done. Arylesterase activity represents phenyl acetate hydrolyzing activity and Paraoxonase activity represents paraoxon-hydrolyzing activity of PON1