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. 2020 Apr 9;7:126. doi: 10.3389/fmed.2020.00126

Table 3.

Multivariate analysis of high overall lipid intake and lipid subtype intake and outcomes in acute pancreatitis.

Lipid Model Adjusted odds ratio
Local complications Persistent organ failure Mortality Moderate-to-severe AP
Lipids (overall) Without obesity 1.21 (0.96–1.53)
P = 0.1
1.21 (0.82–1.8)
P = 0.3
1.15 (0.7–1.88)
P = 0.6
1.26 (1.02–1.55)
P= 0.034
With obesity 1.11 (0.88–1.41)
P = 0.4
1.23 (0.81–1.87)
P = 0.3
1.11 (0.66–1.87)
P = 0.7
1.19 (0.96–1.48)
P = 0.1
SFAs Without obesity 0.98 (0.76–1.26)
P = 0.9
0.87 (0.57–1.32)
P = 0.5
0.76 (0.45–1.28)
P = 0.3
1.06 (0.84–1.3)
P = 0.63
With obesity 1.01 (0.78–1.3)
P = 0.9
0.87 (0.57–1.34)
P = 0.5
0.83 (0.48–1.42)
P = 0.49
1.11 (0.88–1.4)
P = 0.4
UFAs Without obesity 1.53 (1.15–2.05)
P= 0.004
2.1 (1.18–3.74)
P= 0.01
2.37 (1.12–5.03)
P= 0.02
1.42 (1.1–1.85)
P= 0.007
With obesity 1.34 (0.99–1.83)
P = 0.06
2.4 (1.24–4.71)
P= 0.01
2.09 (0.94–4.66)
P = 0.07
1.27 (0.96–1.67)
P = 0.09
MUFAs Without obesity 1.3 (1.03–1.62)
P= 0.025
1.36 (0.92–2)
P = 0.1
1.29 (0.79–2.09)
P = 0.3
1.33 (1.08–1.63)
P= 0.008
With obesity 1.2 (0.95–1.5)
P = 0.1
1.37 (0.91–2.06)
P = 0.1
1.24 (0.75–2.07)
P = 0.4
1.26 (1.02–1.56)
P= 0.035

In the binary logistic regression analysis, the data are reported as adjusted odds ratios (95% confidence intervals) and P-values (reference group: low intake of lipids). Model, the model used for multivariable analysis; Without obesity, the model that included Charlson comorbidity index (cutoff ≥3), sex, alcoholic etiology, and recurrent AP; With obesity, the model that included these same variables plus obesity (body mass index ≥30 kg/m2); SFAs, saturated fatty acids; UFAs, unsaturated fatty acids; MUFAs, monounsaturated fatty acids; AP, acute pancreatitis.

Bold value indicates P-value statistically significant.