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. 2020 Apr 15;20:312. doi: 10.1186/s12885-020-06794-5

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© The Author(s) 2020

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Functional effects of CDR1as inhibition in 293 T cells. a Relative CDR1as expression in different cell lines. b RNA FISH assessment of CDR1as in 293 T and Huh7 cells. Scale bar, 10 μm. c CDR1as knockdown efficiency following siCDR1as transfection. d CDR1as knockdown effects on cell proliferation. e & f CDR1as knockdown effects on cell migration. g CDR1as knockdown effects on cell cycle progression. h Xenograft assessment of tumor size and weight in nude mice using 293 T cells in which CDR1as was knocked down (n = 5). i Tumor volumes from mice in (h)